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This is the strongest piece of evidence in Dihexa's favor, and it is important to frame it accurately: it is a small rodent study in a disease model, not a human trial, and not a measure of cognitive enhancement in healthy people

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Instead, AOD 9604 is used to study downstream metabolic signaling, particularly adipose tissue lipolysis, allowing researchers to isolate lipid metabolism pathways without engaging the broader endocrine GHIGF-1 axis