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These results have implications for neuropsychiatric disorders characterized by accelerated aging molecular profiles, such as major depression

Without Gla on the amino termini of these factors, they no longer bind stably to the blood vessel endothelium and cannot activate clotting to allow formation of a clot during tissue injury

This is because senescent cells can disrupt normal tissue function and composition by secreting pro-inflammatory and matrix-degrading molecules, which is known as the senescence-associated secretory phenotype (SASP) [1719]

They were randomly assigned to receive one of three regimens of teplizumab or a placebo